An example of this are the cases of fracture, as once they occur, the individual does not experience pain. Only later on, after the blood has carried away the synovial fluid poured onto the site, the individual will feel the pain caused by the fracture.

The synovial fluid is present all across the body and does not have a circulation of its own, it relies on specific movements and manual touches to move. The joints are supplied by this protective fluid through the foramens that exist within the articular ligaments. As it does not have its own circulation, it depends on physical movements and the law of gravity to move. This precious fluid, every time a neurologic contraction prevents its presence in a ligamentary base, leads to the formation of calcifications in the neurological lining tissue near the joints. This occurs with some frequency to the stylohyoid ligament. Situations such as this are often encountered by maxillary functional orthopedics experts.

According to studies conducted by Massoprévent, neurocalcemia, which causes deviations to the anatomic and cellular-neurologic patterns, has been confounded with fibromyalgia. This condition causes various pains in the body. People report that on getting up from bed they experience a great deal of sensitivity in the feet and physical rigidity. A slight increase in the hearbeat is also observed, and some anemia is noticed in blood tests, as well as a slight increase in blood calcium levels, with presence of an inflammatory process. Often, an irritation of the genital mucosae, especially the anal one, is present, followed by physical discomfort, insomnia, low performance capacity due to fatigue, followed by drowsiness with neuromuscular irritability, nervousness, irritability and intolerance due to the hardening of neurologic fibers, followed by calcification of their tips.

NEUROCALCEMIA: This neurologic and cellular deviation of anatomic patterns, often confounded by many health care practitioners, results from complications in the anatomic patterns originating from an intracellular infiltrative anomaly.

The complications of neurocalcemia are caused by unbalances in essential minerals such as calcium, but they are often elicited also by invasive agents capable of forming other forms of calcium that are not suitable for the human body.

Such situation eventually causes a calcium buildup inside tissues and blood vessels. It must be remembered that calcium, albeit essential, is a toxic element for the cells when its base is not suitable for human structures. Despite being unique as regards deviations of anatomic patterns in general, irregular calcifications occur concomitantly with several general processes, such as necroses and degenerations, and may be present in any chronic injury.

We said that despite being essential calcium is a toxic element for cells. For this reason, there are diverse complex mechanisms such as the presence of virus, worms, and dietary habits that can keep calcium concentration high or very low in the human body. This allows calcium to play its role as a second messanger in signal translation, participating in major processes as activation, secretion, contraction, exhaustion, and even cell death. For neurocalcemia, therefore, calcium can be considered the main agent of disorders in the cellular, bone, neurologic and anatomic patterns.

The abnormal deposition of minerals and calcium salts - neurocalcemia - may take place in tissues by different ways, as at sites affected by injured tissues. It does not depend on the levels caused by calcium buildup due to circulatory occlusions, forming calcium deposits or calcification of neurologic fibers in the form of calcium and phosphor threads, and in the calcification of the calcium deposit, in which hypercalcemia results from the precipitation of calcium salts in normal tissues. The distinction between the two types of calcification incites discussions and is often regarded as flimsy, as the final physiological aspect is similar, since calcium deposition in healthy tissues causes injury to tissues with some frequency.

Excess calcium may also favor its deposition in the injured tissues, enhancing the deviations of anatomic patterns at previously affected sites. However, the presence of signs of previous injury associated to the greater intensity of calcareous deposition suggests calcification as the main reason for a deviation from optimal anatomic patterns.

The distribution and location of calcium deposits in the human body may also be helpful in the differentiation between one complication and the other. Often, the term "bone demineralization" has been used to define the lack of calcium in the body. However, many times the individual with this condition also presents calcium deposition in muscle tissues and calcification inside the blood vessels, and ligamentary structures near the joints are shown to be calcified as well (neurocalcemia). This characterizes a poor distribution of calcium in the body, as its lack is observed in the bone structures although this mineral is not absent in the body, which indicates poor calcium distribution as the main motive for bone demineralization, and the calcification of the skin and subcutaneous tissue may also take place. In several places around the world, the deposition of calcium salts in soft tissues has been noted, also including the aorta, the kidneys, tendons, articular ligaments and the heart.

NEUROCALCEMIA - Largely, it has been noted that some parasites that inhabit particular vegetables transform their cell functions into an imperative process of vitamin D production, which is the main vitamin for the fixation of calcium in the human body. In the calcified areas, when touched by the hand, nodes of firm, stony or sandy consistency are perceived. In the breast, these nodes are often caused by the presence of neurologic cramps within the upper limbs, which causes a change to the blood flow throughout the mammary region.

Mammary calcification is more frequent than the calcification that occurs inside the blood vessels and is focused on a more specific location. For fibrous connective tissues,such as muscles and articular ligaments, calcification is slower and longer, degenerating the muscle structures, as occurs in the walls of sclerotic vessels, uterine arteries of elderly women, tendons, heart valves, etc.

NEUROCALCEMIA - Calcifications in general also occur in sites of non-absorbed previous necrosis, as in the cases of chronic tendinitis, previous infarctions, around dead parasites and larvae (dead worms are worse than living ones), leading to necrosis of fats in which inflammations occur, or chronic processes that are difficult to resolve. It is quite common to see a prostate inflammation being caused by tendinitis, and the evidence for prostate tendinitis are the cramps in the calves during the sexual intercourse, which makes hard for men to fully consummate the sexual act.

Following the cramps in the calves, the body begins to enhance the calcifications in the ligaments that surround the prostate, leading to prostatitis, among other prostatic disorders. At times, this process is accompanied by other complications such as the buildup of dead parasites, giving rise to pathological calcifications able to deviate the anatomic patterns of the prostate.

NEUROCALCEMIA -The calcium phosphate crystals deposited in the pathological calcifications are similar to those of the bones, these particles from the extracellular matrixes of calcium phosphate have a different composition and activity from those of the human anatomic pattern that originated them.

Usually, the human body produces calcium molecules under optimal conditions and does not need to rely on help from others, such as parasites, which contribute to calcium formation. The absence of calcium in bone structures does not necessarily imply the lack of this mineral in the body, because this condition may occur due to dysfunctions in the production of collagen resulting from the lack of contact on the frontal teeth, since an optimal contact on the frontal teeth is an ongoing stimulus to the thyroid gland, whose function is to regulate several hormones and, especially, the production and distribution of the collagen that fixes calcium to bone structures.

Massoprévent has found this condition in patients with previous history of presence of parasites, triggering an action by the NMD on neurologic fibers, and these fibers, when contracting, are highly likely to calcify as the neurologic defense, when triggered, does not typically cease on its own, depending on specific manual touches. The irregularities in calcium distribution in the organism may cause pain even if there is no calcification of the neurologic fibers.

NEUROCALCEMIA - The contraction of the neurologic microfibers, which causes them to harden, is enough to cause pain, working as a sort of thorn in the musculature. What one perceives, however, with manual touches, is that, in view of calcium dysfunction, the neurologic fibers become calcified and form a kind of fishbone (neurocalcemia) in several organs, blood vessels, connective tissues, and even the musculature.

Many times, via suitable exams, calcifications are found to have occurred in major veins and arteries.

Another important finding is the fact that most of these people present slight anemia, as observed through laboratory tests. The Natural Physical Sciences ascribe this fact to the likelihood for minor blood losses inside the muscle tissues when these are pricked by the calcified neurologic fibers. This pricking by calcified neurologic fibers may occur when one performs a movement.

The Natural Physical Sciences by means of the Massoprévent technique has developed efficacious solutions for neurocalcemia.